- lipid depleted
Animals are unable to take up or store lipids. In C. elegans, this defect results in pale, skinny and arrested larva.
- dye filling defect
Animals exhibit variations in the extent to which one or more sensory neurons associated with sensilla (typically one or more amphids and/or phasmids) take up dye from the environment, compared to controls. In C. elegans, these neurons are commonly assayed by dye-filling with FITC, DiI or DiO.
- M lineage variant
The descendants of the M precursor cell exhibit variations in developmental programs compared to their counterparts in control animals. In C. elegans the M lineage is a postembryonic mesodermal lineage.
- endoreduplication variant
Any variation in the multiple rounds of DNA replication that take place without chromosome condensation, segregation or cytokinesis compared to control. In C. elegans this process is normally seen in intestinal nuclei during each larval lethargus which results in adult intestinal nuclei with 32 copies (32C) of each chromosome(Wormatlas).
- cell cycle arrest
Cells of the animals cease during one of its replicative phases (G1, S, G2, M).
- early exit cell cycle
Cells leave the M phase (mitosis and cytokinesis) at an earlier time than sister or other control cells.
- wing cilia morphology variant
Animals exhibit variations in the structure or organization of the microtuble-based projections that in control animals invaginate individually into the sheath cell of the neuronal sensillum rather than projecting through the socket cell to the outside. In C. elegans, the cilia of each wing cell has its own unique shape and as they are not exposed to the outside, they do not typically take up dyes.
- male M lineage variant
The descendants of the M precursor cell in male animals, exhibit any variation in developmental programs compared to their counterparts in control animals.