male mating efficiency eliminated

Males sire no cross progeny.

hermaphrodite mating efficiency eliminated

Hermaphrodites do not produce cross progeny.

male sterile

X0 animals fail to generate viable cross-progeny.

hermaphrodite sterile

Hermaphrodites fail to generate viable self-progeny and/or cross-progeny.

male mating efficiency reduced

The number of cross progeny sired by males is reduced compared to control.

hermaphrodite mating efficiency reduced

The number of cross progeny produced by hermaphrodites is reduced compared to control hermaphrodites.

excess intestinal cells

Animals contain an excess number of intestinal cells compared to control. In C. elegans, intestinal cells are derived from E-blastomere. The overproduction of intestinal tissues is a result of other blastomeres adopting an E-like fate or excess cell proliferation in the E lineage.

E lineage variant

The descendants of the E blastomere exhibit altered developmental programs compared to their counterparts in control animals.

no Intestine

Embryos fail to generate cells that exhibit intestinal specification. In C. elegans, the E blastomere is the precursor of all the intestinal cells.

organism hypersensitive interstrand cross link agents

Animals exhibit increased sensitivity to interstrand cross-linking agents, such as HN2 (nitrogen mustard) and CDDP (cisplatin) compared to control. In C. elegans, increased sensitivity to these agents is often observed as a decrease in survival compared to control animals after exposure to the agent.