21ur-13263 : 21ur-2896 : 21ur-4405 : 21ur-5298 : hlh-17 : hlh-31 : hlh-32

Gustatory plasticity is restored in hlh-17, hlh-31, hlh-32 animals.

hlh-2 : hlh-2/hlh-3 heterodimer binding site I : hlh-3

A HLH-2 homodimer and HLH- 2/HLH-3 heterodimer bind to wild-type Region B in vitro.

hlh-2 : hlh-2/hlh-3 heterodimer binding site II : hlh-3

A HLH-2 homodimer and HLH- 2/HLH-3 heterodimer bind to wild-type Region B in vitro.

hlh-2 : hlh-2/hlh-3 heterodimer binding site IV : hlh-3

A HLH-2 homodimer and HLH- 2/HLH-3 heterodimer bind to wild-type Region B in vitro.

hlh-2 : hlh-2/hlh-3 heterodimer binding site III : hlh-3

A HLH-2 homodimer and HLH- 2/HLH-3 heterodimer bind to wild-type Region B in vitro.

hlh-3 : ttx-3

As in hlh-16 mutants, ttx-3 expression is lost in around 20% of the AIY lineages in hlh-3 mutant embryos and larvae. In hlh-3; hlh-16 double mutants, ttx-3 expression is lost in nearly all AIY lineages similarly to what is observed in hlh-2 RNAi-treated animals, suggesting that ttx-3 expression may be regulated by two partly redundant bHLH complexes HLH-2:HLH-3 and HLH-2:HLH-16.

Region 'B' : hlh-2 : hlh-3

A HLH-2 homodimer and HLH- 2/HLH-3 heterodimer bind to wild-type Region B in vitro.

Interaction involving hlh-2

"Although hlh-2(bx115) mutants have a normal number of HSN and PHB neurons, hlh-2(bx115); hlh-14(RNAi) animals lack more HSN and PHB neurons than hlh-14(RNAi) animals, showing that a weak hlh-2 mutant can enhance partial hlh-14 loss (Table 4)."

Interaction involving hlh-2

"Although hlh-2(bx115) mutants have a normal number of HSN and PHB neurons, hlh-2(bx115); hlh-14(RNAi) animals lack more HSN and PHB neurons than hlh-14(RNAi) animals, showing that a weak hlh-2 mutant can enhance partial hlh-14 loss (Table 4)."

hlh-29 : med-1

Animals carrying med-1 mutations showed increased hlh-29/hlh-28 activity.