mild hmr-1 RNAi (feeding with diluted bacteria) caused severe epidermal phenotypes and embryonic arrest of ulp-2(gk916250) hypomorph (Thompson et al., 2013), which otherwise developed normally.
"both wild-type and EQ mutant DPY-27::GFP interacted well with MIX-1(SMC-2 homolog). However, DPY-27::GFP co-IPed better with DPY-26 (kleisin subunit of condensin I and DC) compared to DPY-27(EQ)::GFP, suggesting that ATPase mutation affects SMC-kleisin interaction."
"both wild-type and EQ mutant DPY-27::GFP interacted well with MIX-1(SMC-2 homolog). However, DPY-27::GFP co-IPed better with DPY-26 (kleisin subunit of condensin I and DC) compared to DPY-27(EQ)::GFP, suggesting that ATPase mutation affects SMC-kleisin interaction."