"We therefore hypothesized that the
et19 mutant may also be resistant to statins because of an activated mitochondrial stress response. Consistent with this hypothesis,
et19 mutant worms constitutively express high levels of
hsp-60::GFP, a known marker of UPRmt (Figure 2D) (Yoneda et al. 2004; Rauthan et al. 2013)."