rol-3 : rol-6

Animals were right-rollers as L3's, but squattish nonrollers as L4's and adults.

F53A9.8 : Heat-Killed Staphylococcus Aureus : Y65B4BR.1 : clec-52 : clec-60 : clec-71 : cpr-2 : exc-5 : fmo-2 : ilys-3 : lys-5

"...We measured gene induction during exposure to live S. aureus, which causes pathogenesis, or heat-killed S. aureus, which does not (Figures 3G-I and S4C, E). Unexpectedly, all 10 biomarker genes were induced at least equally well on heat-killed S. aureus as on live S. aureus (Figure 6A)."

unc-33 : unc-44

UNC-33(S) accumulated at the nerve ring of control animals, and unc-44 mutants induced more diffuse distribution of UNC-33(S).

bacteria Staphylococcus aureus : irg-4

F08G5.6 was specifically induced by S. aureus-mediated infections.

cholesterol starvation : sams-1

SAMS-1 (S-adenosylmethionine synthetase) is down-regulated under cholesterol starvation.

cdkl-1 : dyf-5

Loss of cdkl-1 had no effect on the truncated cilia phenotypes in either ASH or AWA in dyf-5(XS) animals (Figure 1A-D), suggesting that similar to observations in ADL (Park et al., 2021), CDKL-1 is also unlikely to act in a linear pathway with DYF-5 to regulate cilia length in ASH or AWA.

daf-3 : daf-4

daf-3's transcriptional repressor activity is negatively regulated by daf-4.

Interaction involving unc-33

UNC-33 isoform M and S interacts each other or by itself.

atf-7 : bacteria Staphylococcus aureus

ATF-7 mRNA was downregulated during the subsequent infection of S. aureus.

atp-3 : egl-15

Query-target gene pairs were tested for interaction by feeding target gene RNAi to worms with a mutation in the query gene. Each query-target pair and its two controls were tested in at least three rounds.|Target genes were classified as either belonging to LGIII (L) or to signalling pathways (S) or to both (L,S): S