- lev-1 : nlp-3 : unc-29 : unc-38
This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.
- lev-1 : nlp-7 : unc-29 : unc-38
This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.
- lev-1 : nlp-12 : unc-29 : unc-38
This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.
- lev-1 : nlp-2 : unc-29 : unc-38
This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.
- lev-1 : nlp-15 : unc-29 : unc-38
This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.
- Tunicamycin : dnj-27
Treatment with 10, 20 and 30 μg/ml tunicamycin resulted in a strong increase of GFP fluorescence of Pdnj-27::GFP reporter.
- MG132 : quercetin
"Additionally, inhibiting proteasomal degradation by use of the specific proteasome inhibitor MG132 (200 μM) completely blocked the rescuing effect of quercetin with respect to survival (Fig. 7D)."
- molo-1 : unc-29
"MOLO-1 and L-AChRs specifically interact in a physical complex."
- molo-1 : unc-63
"MOLO-1 and L-AChRs specifically interact in a physical complex."