car-1 : cey-2

"CAR-1 associates with the Y-box containing protein CEY-2."

egl-5 : vab-3

In contrast to wild-type, vab-3::gfp expression does not initiate in the tail of egl-5(n486) mutants, and is absent from the B and Y cell lineages.

cgh-1 : gld-2

In a strong gld-2 loss-of-function allele(q497) CGH-1 was appropriately upregulated at meiosis entr y and appeared to be localized normally through the pachytene stage, but expression of both CGH-1 and PGL-1 was lost more proximally, in the abnormal gametes that are produced.

cgh-1 : gld-2

In a strong gld-2 loss-of-function allele(q497) CGH-1 was appropriately upregulated at meiosis entr y and appeared to be localized normally through the pachytene stage, but expression of both CGH-1 and PGL-1 was lost more proximally, in the abnormal gametes that are produced.

cgh-1 : gld-2

In a strong gld-2 loss-of-function allele(q497) CGH-1 was appropriately upregulated at meiosis entr y and appeared to be localized normally through the pachytene stage, but expression of both CGH-1 and PGL-1 was lost more proximally, in the abnormal gametes that are produced.

cgh-1 : gld-2

In a strong gld-2 loss-of-function allele(q497) CGH-1 was appropriately upregulated at meiosis entr y and appeared to be localized normally through the pachytene stage, but expression of both CGH-1 and PGL-1 was lost more proximally, in the abnormal gametes that are produced.

cgh-1 : gld-2

In a strong gld-2 loss-of-function allele(q497) CGH-1 was appropriately upregulated at meiosis entr y and appeared to be localized normally through the pachytene stage, but expression of both CGH-1 and PGL-1 was lost more proximally, in the abnormal gametes that are produced.

mec-4 : mec-6

The mec-6 gene is required for the punctate distribution of MEC-4. When mec-4::yfp and promoter mec-4::cfp were injected into mec-6(u3) animals, the expression of the promoter mec-4::cfp fusion was unchanged, but the punctate expression from mec-4::y fp was undetectable.

ain-1 : ain-2 : irg-4 : sod-3

"we examined mRNA levels of a subset of these genes in ain-2[reduction-of-function (rf)];ain-1(lf) double mutants in which miRISC activity is strongly and specifically reduced (Zhang et al., 2007). We found a significant upregulation in eight of the nine AIN-2-associated, dcr-1- and pathogen-responsive genes we tested (we refer to these genes as miRISC-regulated pathogen-response [MRPR] genes hereafter) (Figure 4B). As controls, we examined the expression of ges-1 (an intestine-specific, pathogen nonresponsive gene), F08G5.6 (a pathogen-responsive gene encoding mRNAs that were not detected in the miRISC), and sod-3 (a general stressinduced gene) and found no change in the expression of these genes in ain-2(rf);ain-1(lf) double mutants."

C49G7.10 : F31F7.1 : K09D9.1 : ain-1 : ain-2 : clc-19 : clec-63 : hbl-1 : ncr-1 : tsp-1

"we examined mRNA levels of a subset of these genes in ain-2[reduction-of-function (rf)];ain-1(lf) double mutants in which miRISC activity is strongly and specifically reduced (Zhang et al., 2007). We found a significant upregulation in eight of the nine AIN-2-associated, dcr-1- and pathogen-responsive genes we tested (we refer to these genes as miRISC-regulated pathogen-response [MRPR] genes hereafter) (Figure 4B). As controls, we examined the expression of ges-1 (an intestine-specific, pathogen nonresponsive gene), F08G5.6 (a pathogen-responsive gene encoding mRNAs that were not detected in the miRISC), and sod-3 (a general stressinduced gene) and found no change in the expression of these genes in ain-2(rf);ain-1(lf) double mutants."