lev-1 : nlp-3 : unc-29 : unc-38

This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.

lev-1 : nlp-7 : unc-29 : unc-38

This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.

lev-1 : nlp-12 : unc-29 : unc-38

This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.

lev-1 : nlp-2 : unc-29 : unc-38

This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.

lev-1 : nlp-15 : unc-29 : unc-38

This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.

daf-16 : daf-2

daf-16 RNAi suppressed the long lifespan of daf-2(e1370).

age-1 : daf-16

daf-16 RNAi suppressed the long lifespan of age-1(hx546).

daf-16 : mir-239.1 : mir-239.2

daf-16(RNAi) completely abolished the long life span of mir-239(nDf62) mutants, showing that the long lived phenotype of mir-239 mutants depends on the presence of daf-16.

age-1 : daf-18

daf-18(RNAi) suppressed the long-lived phenotype of age-1(hx546)

age-1 : pdp-1

pdp-1(RNAi) suppressed the long-lived phenotype of age-1(hx546)