daf-2 : let-60

let-60(n1046gf) did not suppress the dauer-constitutive phenotype of daf-2(e1370) (J.Apfeld and C. Kenyon, unpublished data).

bar-1 : lin-39

Authors expressed a lin-39 Hox cDNA driven from a heat-shock promoter (Hunter and Kenyon, 1995) in bar-1(ga80) mutants, and found that heat-shock-induced lin-39 Hox expression rescued the F fate defect (fusion with the large hypodermal syncytium) of P3.p and P4.p (Table 2).

daf-11 : trx-1

At 25 deg C, but enhances the Daf-C at 15 deg C.

daf-11 : trx-1

At 15 deg C but suppresses the Daf-C phenotype of sa195 at 25 deg C.

acdh-11 : high temperature

Growth at 25 C as opposed to 20 C or 15 C caused marked up regulation of Pacdh-11::GFP predominantly in the intestine.

daf-2 : trx-1

No animals were dauer at 15 deg C and no enhancement or suppression of the Daf-C phenotype was observed at 25 deg C.

daf-2 : trx-1

No animals were dauer at 15 deg C and no enhancement or suppression of the Daf-C phenotype was observed at 25 deg C.

daf-2 : trx-1

No animals were dauer at 15 deg C and no enhancement or suppression of the Daf-C phenotype was observed at 20 or 25 deg C.

egl-20 : mab-5

"In the absence of any hs-egl-20, the QL and QR descendants are unable to express mab-5::lacZ in egl-20(n585) animals (top panels). After an intermediate dose of hs-egl-20 (10 min pulse), the QL descendants have turned on mab-5::lacZ expression, whereas many of the QR descendants have not (middle panels). We never observed animals in which mab-5::lacZ was expressed in the QR but not QL descendants. At high doses of hs-egl-20 (30 min pulse), mab-5::lacZ is expressed in both QL and QR descendants (bottom panels). This mab-5::lacZ fusion is expressed in all L1 cells in which mab-5 is predicted to function genetically(Salser and Kenyon, 1992)."

lit-1 : mom-2

100% of embryos lack endoderm in double mutants at 15 deg C and 25 deg C, whereas lit-1 mutants only exhibit this phenotype at 25 deg C and mom-2 mutants exhibit this phenotype only partially at 22 deg C.