ced-10 : mig-2

In most animals only P5.p or P7.p descendants showed migration defect, but in 45 percent both P5.p and P7.p descendants showed Mig defects.

Cbr-lin-39 : Cbr-pry-1

The small nucleus phenotype of P7.p, P8.p, and P11.p was not suppressed by bh20 (data not shown).

ced-10 : mig-2

In most animals only P5.p or P7.p descendants showed migration defect, but in 45 percent both P5.p and P7.p descendants showed Mig defects. Many cells that should have divided along the transverse axis in P6.p and P7.p descendants instead divided along the longitudinal axis.

lin-1 : lip-1

In the N2 worm shown, the expression of lip-1::gfp is upregulated in P5.p (strongly) and P7.p descendants, and downregulated in P6.p descendants. In the lin-1(n304) mutant, expression is strongly upregulated in P6.p descendants but not in P5.p or P7.p descendants.

let-60 : lin-1

"In 72% of lin-15(rf) (n = 18) and 68% of let-60(gf) animals (n = 34), LIP-1::GFP was up-regulated in P3.p, P4.p, or P8.p in addition to P5.p and P7.p (Fig. 2F)."

lin-1 : lip-1

In the N2 worm shown, the expression of lip-1::gfp is upregulated in P5.p (strongly) and P7.p descendants, and downregulated in P6.p descendants. In the lin-1(n304) mutant, expression is strongly upregulated in P6.p descendants but not in P5.p or P7.p descendants.

lin-17 : lin-18

Double mutants were 100%P-Rvl n=40, no P-value was assigned.

mab-5 : ref-1

In ref-1; mab-5(e1751), P1.p and P2.p often remained unfused.

let-23 : sft-4

sft-4 RNAi caused normal localization in P6.p but persistent expression in P7.p.

egl-5 : ref-1

The introduction of an egl-5 mutation into ref-1 animals caused a 35% decrease in the P9.p and P10.p cell fusion defect(P=0.001).