cyd-1 : elt-2

"We have used reporter gene expression to assay directly the divisions of the mesoblast cellM (hlh-8::GFP, Harfe et al., 1998) and the intestinal nuclei (elt-2::GFP, Fukushige et al., 1998). These two sets of divisions are completely blocked following cyd-1(RNAi)(Fig. 5B)"

elt-2 : itr-1

"we investigated the fate of the E lineage by using an elt-2::gfp transgene (Fukushige et al., 1999)... Overall, expression of ELT-2::GFP was observed in only 35.1% of embryos in super-sponge worms, compared with 95.9% for the control sponge (Figure 5, L-O)."

cyd-1 : hlh-8

"We have used reporter gene expression to assay directly the divisions of the mesoblast cellM (hlh-8::GFP, Harfe et al., 1998) and the intestinal nuclei (elt-2::GFP, Fukushige et al., 1998). These two sets of divisions are completely blocked following cyd-1(RNAi)(Fig. 5B)"

cdc-25.1 : elt-2

"we administered cdc-25.1 RNAi to young adult hermaphrodites by the bacterial feeding method (Timmons et al., 2001) and examined their F1 progeny... We used two strains of C. elegans, JR1838, which carries an elt-2::GFP transgene (Fukushige et al., 1998)... Using the C. elegans strain JR1838, ~30% of the dying embryos had no cells expressing the intestinal marker (Figure 1K and 1L)"

cdc-25.1 : elt-2

"we screened for mutants that would phenocopy this extra intestinal cell phenotype, using the intestinal-specific reporter elt-2::GFP (Fukushige et al., 1998). 10,320 haploid genomes were screened and although several mutants with fewer intestinal cells were isolated, we have identified only one mutant with extra intestinal cells. This mutant, rr31, has 57+/-4 intestinal nuclei at the adult stage, or approximately twice the wild-type complement (Fig. 1; Table 1). "

cbp-1 : elt-2

"RNAi inhibition of CBP-1 induces ectopic expression of HLH-2::GFP in non-neuronal cells (Figure 4B, compare panels a and b; Shi and Mello, 1998), a promoter who's activity is restricted to neurons late in C. elegans embryogenesis (Krause et al., 1997). In contrast, inhibition of CBP-1 by RNAi eliminated the expression of HLH-1::GFP and ELT-2::GFP (Figure 4B, compare panels e and h with d and g), promoters that are active in muscle and endodermal cells, respectively (Krause et al., 1990; Fukushige et al., 1998)."

cbp-1 : hlh-1

"RNAi inhibition of CBP-1 induces ectopic expression of HLH-2::GFP in non-neuronal cells (Figure 4B, compare panels a and b; Shi and Mello, 1998), a promoter who's activity is restricted to neurons late in C. elegans embryogenesis (Krause et al., 1997). In contrast, inhibition of CBP-1 by RNAi eliminated the expression of HLH-1::GFP and ELT-2::GFP (Figure 4B, compare panels e and h with d and g), promoters that are active in muscle and endodermal cells, respectively (Krause et al., 1990; Fukushige et al., 1998)."

cbp-1 : hlh-2

"RNAi inhibition of CBP-1 induces ectopic expression of HLH-2::GFP in non-neuronal cells (Figure 4B, compare panels a and b; Shi and Mello, 1998), a promoter who's activity is restricted to neurons late in C. elegans embryogenesis (Krause et al., 1997). In contrast, inhibition of CBP-1 by RNAi eliminated the expression of HLH-1::GFP and ELT-2::GFP (Figure 4B, compare panels e and h with d and g), promoters that are active in muscle and endodermal cells, respectively (Krause et al., 1990; Fukushige et al., 1998)."

elt-2 : pho-1

"To demonstrate that ELT-2 is indeed necessary for in vivo expression of pho-1, elt-2(RNAi) was performed in the strain containing integrated copies of the 1-kb pho-1 promoter fused to a GFP/lacZ reporter... Injection of elt-2 double-stranded RNA into transgenic pho-1::GFP/lacZ hermaphrodites produces highly penetrant (100% in most replicates) arrest of F1 larvae, all of which show the distinctive Gob (gut-obstructed) phenotype seen with the elt-2 knockout (Fukushige et al., 1998, 2003). In these arrested larvae, β-galactosidase (and GFP) reporter gene expression is almost completely abolished."