plx-1 : unc-60

In wild type, lin-32p::mRFP::unc-60A 3'UTR was intensely detected in every R(n).a + p. However, in plx-1 mutants, R(1).a + p, R(2).a + p and R(5).a + units displayed pronounced decreases in expression of mRFP.

egl-5 : ref-1

The introduction of an egl-5 mutation into ref-1 animals caused a 35% decrease in the P9.p and P10.p cell fusion defect(P=0.001).

smp-1 : smp-2 : unc-60

In wild type, lin-32p::mRFP::unc-60A 3'UTR was intensely detected in every R(n).a + p. However, in smp-1 smp-2 mutants, R(1).a + p, R(2).a + p and R(5).a + units displayed pronounced decreases in expression of mRFP.

daf-16 : kaempferol : myricetin : naringenin : quercetin

All tested flavonoids increased nuclear localization of DAF-16 in gut epithelial cells (myricetin p = 0.0469, quercetin p = 0.004, kaempferol p = 0.04 and naringenin p = 0.0455, Student's t-test), demonstrating a reduced DAF-16 phosphorylation status after flavonoid treatment.

Interaction involving pgl-3

"PGL dimers are a key building block for P-granule assembly."

Interaction involving pgl-3

"PGL dimers are a key building block for P-granule assembly."

glr-1 : klp-4

klp-4(pz19) mutant animals expressing an integrated GLR- 1::GFP transgene (nuIs25) exhibit a 37% decrease in GLR-1::GFP puncta intensity (p<0.001) and a 21% decrease in puncta density (p<0.001) compared to wild type controls. klp-4(tm2114) mutants exhibit a 39% decrease in GLR-1::GFP puncta intensity (p<0.001) and a 27% decrease in puncta density (p<0.001) compared to wild type controls.

Interaction involving pgl-1

"PGL dimers are a key building block for P-granule assembly."

C05B10.17 : lip-1 : pry-1

A significant increase in P7.p induction was observed in addition to egl-17::GFP (ayIs4; secondary fate marker) expression in induced P7.p.

let-23 : tcl-2

let-23(sy97) and tcl-2(mh15) combine to produce a more-than-additive penetrance of P12.p to P11.p cell transformation defect