In contrast to what has been reported in mice, we observed elevated embryonic lethality in brc-1 and brd-1 null alleles [brc-1(xoe4), brd-1(xoe18) (Li et al. 2023; Li et al. 2018)], in combination with either hsr-9(ok759) (Ryu et al. 2013) or a new putative null allele hsr-9(xoe17) (Figure 1A).
In contrast to what has been reported in mice, we observed elevated embryonic lethality in brc-1 and brd-1 null alleles [brc-1(xoe4), brd-1(xoe18) (Li et al. 2023; Li et al. 2018)], in combination with either hsr-9(ok759) (Ryu et al. 2013) or a new putative null allele hsr-9(xoe17) (Figure 1A).
RNAi against spn-2 partially suppressed the ray 1 phenotype in plx-1 mutants. Ray 1 is frequently displaced anteriorly, sometimes outside the fan, in plx-1 mutants.
In contrast to what has been reported in mice, we observed elevated embryonic lethality in brc-1 and brd-1 null alleles [brc-1(xoe4), brd-1(xoe18) (Li et al. 2023; Li et al. 2018)], in combination with either hsr-9(ok759) (Ryu et al. 2013) or a new putative null allele hsr-9(xoe17) (Figure 1A).
In contrast to what has been reported in mice, we observed elevated embryonic lethality in brc-1 and brd-1 null alleles [brc-1(xoe4), brd-1(xoe18) (Li et al. 2023; Li et al. 2018)], in combination with either hsr-9(ok759) (Ryu et al. 2013) or a new putative null allele hsr-9(xoe17) (Figure 1A).
RNAi against gcn-1 in plx-1 mutants suppressed the ray 1 phenotype of plx-1. Ray 1 is frequently displaced anteriorly, sometimes outside the fan, in plx-1 mutants.
On the other hand, a hypomorphic brc-1 allele, brc-1(tm1145) (Li et al. 2018), in combination with hsr-9(xoe17) did not result in elevated embryonic lethality (Figure 1A).