Worms co-expressing BAX inhibitotor (BI) showed healthy dopamine neurons with normal neuron morphology and continuity, similar to that of GFP control animals.
Worms co-expressing BAX inhibitor (BI) and BAX had a food sensing ability comparable with that of non-BAX worms, unlike worms expressing BAX alone, which exhibited no slowing in response to food.
In a strong gld-2 loss-of-function allele(q497) CGH-1 was appropriately upregulated at meiosis entr y and appeared to be localized normally through the pachytene stage, but expression of both CGH-1 and PGL-1 was lost more proximally, in the abnormal gametes that are produced.
In a strong gld-2 loss-of-function allele(q497) CGH-1 was appropriately upregulated at meiosis entr y and appeared to be localized normally through the pachytene stage, but expression of both CGH-1 and PGL-1 was lost more proximally, in the abnormal gametes that are produced.
In a strong gld-2 loss-of-function allele(q497) CGH-1 was appropriately upregulated at meiosis entr y and appeared to be localized normally through the pachytene stage, but expression of both CGH-1 and PGL-1 was lost more proximally, in the abnormal gametes that are produced.
In a strong gld-2 loss-of-function allele(q497) CGH-1 was appropriately upregulated at meiosis entr y and appeared to be localized normally through the pachytene stage, but expression of both CGH-1 and PGL-1 was lost more proximally, in the abnormal gametes that are produced.
In a strong gld-2 loss-of-function allele(q497) CGH-1 was appropriately upregulated at meiosis entr y and appeared to be localized normally through the pachytene stage, but expression of both CGH-1 and PGL-1 was lost more proximally, in the abnormal gametes that are produced.
The mec-6 gene is required for the punctate distribution of MEC-4. When mec-4::yfp and promoter mec-4::cfp were injected into mec-6(u3) animals, the expression of the promoter mec-4::cfp fusion was unchanged, but the punctate expression from mec-4::y fp was undetectable.