- mam-1 [Browse genome (BioProject PRJNA13758)] [Search on AGR]
Caenorhabditis elegans Predicted to enable transmembrane signaling receptor activity. Predicted to be involved in regulation of T cell activation and regulation of T cell migration. Predicted to be located in membrane.
- Gm40600 [Search on AGR]
Mus musculus PHENOTYPE: Homozygous knockout and transgenic overexpression indicate that the gene product indirectly promotes B cell response to T cell-dependent antigens via CD4+ T cell activation and directly suppresses B cell response to T cell-independent antigens. [provided by MGI curators]
- Bmf [Search on AGR]
Mus musculus PHENOTYPE: Mice homozygous for targeted knockout mutations show enlarged spleen, increased B cells and CD8-positive T cells, decreased B cells and T cells apoptosis, vagina atresia and hydrometrocolpos. [provided by MGI curators]
- Lax1 [Search on AGR]
Mus musculus PHENOTYPE: Homozygous null mice exhibit spontaneous germinal center formation and increased IgG1, IgG2a, and IgE levels. T and B cells are hyperresponsive upon engagement of T or B AgRs. [provided by MGI curators]
- Zbtb1 [Search on AGR]
Mus musculus PHENOTYPE: Mice homozygous for an ENU induced mutation exhibit abnormal thymus, B cell, and T cell differentiation, and reduced numbers of T, B, and NK cells in the spleen. [provided by MGI curators]
- CARD11-AS1 [Search on AGR]
Homo sapiens ASSOCIATED WITH Asthma; asthma; B-CELL EXPANSION WITH NFKB AND T-CELL ANERGY
- Icos [Search on AGR]
Mus musculus PHENOTYPE: Mice homozygous for disruptions in this gene show reduced basal IgG1 levels and impaired interactions between T and B cells. T cell dependent B cell isotype switching was impaired as well. [provided by MGI curators]
- Rftn1 [Search on AGR]
Mus musculus PHENOTYPE: Mice homozygous for null mutations have defects in T cell activation, T cell help to B cells, and Th17 differentation. [provided by MGI curators]