The response of the nematode C. elegans to Y. pestis infection was evaluated by gene expression profiling. A synchronized population of nematodes were exposed to Y. pestis KIM5 for 24h. Transcript levels from Y. pestis-treated animals were compared with animals maintained on relatively nonpathogenic E. coli OP50 for 24h.
A quantitative shotgun proteomics approach using 15N-based metabolic labeling was conducted at 20, 40, and 60 h during development starting with L1-larvae of N2 animals.
A quantitative shotgun proteomics approach using 15N-based metabolic labeling was conducted at 20, 40, and 60 h during development starting with L1-larvae of N2 animals.
A quantitative shotgun proteomics approach using 15N-based metabolic labeling was conducted at 20, 40, and 60 h during development starting with L1-larvae of N2 animals.
A quantitative shotgun proteomics approach using 15N-based metabolic labeling was conducted at 20, 40, and 60 h during development starting with L1-larvae of N2 animals.
A quantitative shotgun proteomics approach using 15N-based metabolic labeling was conducted at 20, 40, and 60 h during development starting with L1-larvae of N2 animals.
A quantitative shotgun proteomics approach using 15N-based metabolic labeling was conducted at 20, 40, and 60 h during development starting with L1-larvae of N2 animals.
A quantitative shotgun proteomics approach using 15N-based metabolic labeling was conducted at 20, 40, and 60 h during development starting with L1-larvae of pept-1(lg601) animals.
A quantitative shotgun proteomics approach using 15N-based metabolic labeling was conducted at 20, 40, and 60 h during development starting with L1-larvae of pept-1(lg601) animals.
A quantitative shotgun proteomics approach using 15N-based metabolic labeling was conducted at 20, 40, and 60 h during development starting with L1-larvae of pept-1(lg601) animals.